Sample Research Paper on Multiple Sclerosis - Essay.
Multiple Sclerosis The primary objective of this paper is to raise fundamental questions in regards to multiple sclerosis, and to explore possibilities that attempt to answer these inquiries. Second, the prospective outcome is to provide a solid knowledge base for which my peers may begin to understand the relationship between multiple sclerosis and neurobiology and behavior.
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In my paper I will explain what MS is, who gets MS, what MS has to do with the metabolism, some new techniques being used to pinpoint genetic factors, what some of the symptoms of MS is, and some treatments for MS. Multiple Sclerosis Multiple sclerosis (MS) is a progressive disabling illness that affects nerve cells in the brain and spinal.
Multiple Sclerosis is an area of ever expanding research and escalating publications.Multiple Sclerosis and Related Disorders is a wide ranging international journal supported by key researchers from all neuroscience domains that focus on MS and associated disease of the central nervous system.The primary aim of this new journal is the rapid publication of high quality original research in the.
Editorial Board Multiple Sclerosis Journal is a peer-reviewed international journal that focuses on all aspects of multiple sclerosis, neuromyelitis optica and other related autoimmune diseases of the central nervous system. The journal for your research in the following areas.
RESEARCH PAPER MUltiple Sclerosis and Extract of Cannabis: results of the MUSEC trial John Peter Zajicek,1 Jeremy C Hobart,1 Anita Slade,1 David Barnes,2 Paul G Mattison,3 on behalf of the MUSEC Research Group ABSTRACT Objective Multiple sclerosis (MS) is associated with chronic symptoms, including muscle stiffness, spasms, pain and insomnia.
Multiple Sclerosis (MS) MS is a disorder which creates communication difficulties in nerve cells in the brain and spinal cord; in their ability to send action potentials along axons. MS is defined as the demyelination and scarring of the axons causing damage to the myelin sheaths around the axons as a result of an autoimmune response.